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Sas 412/8/2023 ![]() ![]() LDL are catabolized mainly in hepatocytes and other cells after receptor-mediated Remainder are converted to LDL by further removal of triglycerides mediated by hepatic Lipoproteins), some of which undergo endocytosis directly into hepatocytes. Depletion of triglycerides produces VLDL remnants (or intermediate-density Storage in adipose tissue and for oxidation in tissues such as cardiac and skeletal also hydrolyzed by the lipoprotein lipase (LPL) system (yielding free fatty acids for secreted by liver and export triglycerides to peripheral tissue the lipoprotein lipase (LPL) system remove triglycerides from the chylomicrons inĮxtrahepatic tissues via hydrolysis (removal leads to smaller particle size) formed in the intestine and carry triglycerides of dietary origin, unesterified cholesterol,Īnd cholesteryl esters into the bloodstream Hence, control of triglycerides can prevent life-threatening diseases Major clinical sequelae (consequential condition) of hyperlipidemias are acute pancreatitis and atherosclerosis. O Hyperlipemia – increased levels of triglycerides O Hyperlipoproteinemias or Hyperlipidemias – metabolic disorders that involveĮlevations inany lipoprotein species (lipoproteins are plasma lipid complexes)
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